"Do not go where the path may lead, go instead where there is no path and leave a trail" Ralph Waldo Emerson
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Background:Breast milk is a remarkable biological product that, through millions of years of evolution, has evolved to make babies healthy, and define mammals as the dominant animals on this planet. It contains the specified nutrition, immunological factors, and microbes necessary for establishment on the gut microbiome, immune, and neurological development. Health benefits of human milk have long been known, but the importance of the mother's skin microbiota transferred during breastfeeding influencing these health effects is just being discovered.
We on the forefront of this new frontier of research where we are just now understanding that there are 100,000 times the genes in the bacterial microbiome than your own cells that are influencing the very way you think, metabolize food, and genetic expression of disease- It’s who you are! There is a narrow window during the first 3 years of life where the establishment of your signature gut microbiome can be influenced and optimized through the increased volume of human milk. Preterm infants who do not recieve human milk suffer more from consuquences of dysbiosis such as necrotizing enterocolotis, sepsis, and often death. Dysbiosis is associated with the two major killers in preterm infancy: necrotizing enterocolitis and late onset sepsis. Affecting >4000 preterm infants yearly in the United States alone, necrotizing enterocolitis remains the most devastating gastrointestinal disease for preterm infants with a mortality rate of nearly 33% and costs the U.S. healthcare system >$1 billion annually alone. Because a modifiable factor shown to ameliorate the devastating effects of dysbiosis in babies is breast milk, and the scientific community is uncovering the importance of skin-to- skin contact and microbial transfer in the establishment of the life long signature microbiome, a way to increase intake of human milk volume and breastfeeding is crucial, particularly in the preterm infant populaton. |